Abstract

In patients with sarcoidosis, dysregulated calcium metabolism is one of the frequently observed complications. However, little attention has been paid to abnormal phosphate metabolism. Herein we present the case of a 42-year-old Japanese man with renal sarcoidosis who developed acute kidney injury due to hypercalcemia and nephrolithiasis. Laboratory data showed hypercalcemia with a normal serum phosphate level and high serum 1,25-hydroxyvitamin D3, fibroblast growth factor 23 (FGF23) and calciprotein particle (CPP) levels. After treatment with oral prednisone and bisphosphonate, the laboratory abnormalities and renal dysfunction were resolved. Thus increases in FGF23 and CPP may indicate disturbed phosphate metabolism in renal sarcoidosis.

Details

Title
Calciprotein particles and fibroblast growth factor 23 contribute to the pathophysiology of hypercalcemia in a patient with renal sarcoidosis
Author
Iwazu, Yoshitaka 1 ; Kuro-o, Makoto 2 ; Miura, Yutaka 2 ; Takeda, Shin-ichi 3 ; Yamada, Toshiyuki 4 ; Nagata, Daisuke 5 

 Department of Nephrology, Jichi Medical University, Shimotsuke, Tochigi, Japan; Department of Clinical Laboratory Medicine, Tochigi Medical Center Shimotsuga, Jichi Medical University, Shimotsuke, Tochigi, Japan; Department of Medicine, Division of Nephrology, Jichi Medical University, Shimotsuke, Tochigi, Japan 
 Division of Anti-aging Medicine, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan 
 Department of Nephrology, Jichi Medical University, Shimotsuke, Tochigi, Japan; Department of Medicine, Division of Nephrology, Jichi Medical University, Shimotsuke, Tochigi, Japan 
 Department of Clinical Laboratory Medicine, Tochigi Medical Center Shimotsuga, Jichi Medical University, Shimotsuke, Tochigi, Japan 
 Department of Medicine, Division of Nephrology, Jichi Medical University, Shimotsuke, Tochigi, Japan 
Pages
421-423
Publication year
2021
Publication date
Jan 2021
Publisher
Oxford University Press
ISSN
20488505
e-ISSN
20488513
Source type
Scholarly Journal
Language of publication
English
ProQuest document ID
3169590362
Copyright
© The Author(s) 2019. Published by Oxford University Press on behalf of ERA-EDTA. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.