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Abstract

Simple Summary

Tryptophan (Trp) is an essential amino acid that must be acquired exclusively through dietary intake. The metabolism of tryptophan plays a critical role in maintaining immune homeostasis and tolerance, as well as in preventing excessive inflammatory responses. Tryptophan-2,3-dioxygenase (TDO2) serves as one of the pivotal rate-limiting enzymes in the first step of tryptophan metabolism. Dysregulation of TDO2 expression has been found in various digestive system diseases, including digestive system cancers and inflammatory digestive system diseases. Consequently, TDO2 has garnered increasing recognition as a promising therapeutic target for digestive system diseases in recent years, attracting growing attention. This review elucidates the mechanisms by which TDO2 functions within the tryptophan metabolic pathway, its role in digestive system diseases, and recent advancements in TDO2 inhibitor research. The objective is to offer novel insights and a comprehensive theoretical foundation for future investigations in tryptophan metabolism.

Details

1009240
Title
Tryptophan-2,3-Dioxygenase as a Therapeutic Target in Digestive System Diseases
Publication title
Biology; Basel
Volume
14
Issue
3
First page
295
Publication year
2025
Publication date
2025
Publisher
MDPI AG
Place of publication
Basel
Country of publication
Switzerland
Publication subject
e-ISSN
20797737
Source type
Scholarly Journal
Language of publication
English
Document type
Journal Article
Publication history
 
 
Online publication date
2025-03-15
Milestone dates
2025-01-22 (Received); 2025-03-12 (Accepted)
Publication history
 
 
   First posting date
15 Mar 2025
ProQuest document ID
3181354370
Document URL
https://www.proquest.com/scholarly-journals/tryptophan-2-3-dioxygenase-as-therapeutic-target/docview/3181354370/se-2?accountid=208611
Copyright
© 2025 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.
Last updated
2026-01-19
Database
ProQuest One Academic