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Abstract

Neurosyphilis (NS) is a chronic central nervous system infection caused by Treponema pallidum. Owing to its diverse clinical manifestations and the limited sensitivity of current diagnostic methods, NS is difficult to diagnose. Understanding the molecular mechanisms of NS and identifying reliable biomarkers are essential for improving diagnostic and therapeutic strategies. This study employed Mendelian randomization (MR) analysis to explore the causal relationships among protein ratio quantitative trait loci (rQTLs), cerebrospinal fluid (CSF) metabolites, and syphilis risk at various stages. The results revealed that several rQTLs, including CD46/TNFRSF14 and TBC1D23/TBC1D5, were closely associated with syphilis risk, whereas others, such as BANK1/HEXIM1 and GOPC/HEXIM1, exhibited protective effects. Mediation analysis further identified key CSF metabolites, such as N-acetyltaurine and bilirubin, as important mediators linking rQTLs and syphilis progression. Through integrated analysis of cis-proteins from rQTLs and transcriptomic data from CD4 + T-cells of NS patients, METAP2 was identified as a key biomarker in NS, with the potential mechanisms elucidated. Importantly, T. pallidum may inhibit CD4 + T-cell proliferation by modulating METAP2, thereby accelerating disease progression. These findings offer new insights into the pathogenesis of NS and highlight METAP2 as a potential biomarker, laying a foundation for improving diagnostic and therapeutic strategies.

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Title
Treponema pallidum inhibits CD4+ T-cell proliferation through METAP2: insights from Mendelian randomization analysis
Author
Liu, Zhaoping 1 ; Zhang, Xiaohong 2 ; Lin, Ting 3 ; Ding, Xuan 2 ; Yu, Han 2 ; Yao, Jiangchen 2 ; Gao, Ke 2 ; Wu, Yimou 4 ; Zhao, Feijun 5 

 University of South China, Department of Clinical Laboratory Medicine, Institution of Microbiology and Infectious Diseases, Hunan Province Clinical Research Center for Accurate Diagnosis and Treatment of High-incidence Sexually Transmitted Diseases, The First Affiliated Hospital, Hengyang Medical School, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918) 
 University of South China, MOE Key Lab of Rare Pediatric Diseases&Institute of Pathogenic Biology and Key Laboratory of Special Pathogen Prevention and Control of Hunan Province, Hengyang Medical College, Hengyang City, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918) 
 University of South China, School of Public Health, Hengyang Medical School, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918) 
 University of South China, MOE Key Lab of Rare Pediatric Diseases&Institute of Pathogenic Biology and Key Laboratory of Special Pathogen Prevention and Control of Hunan Province, Hengyang Medical College, Hengyang City, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918); University of South China, Department of Clinical Laboratory Medicine, Institution of Microbiology and Infectious Diseases, Hunan Province Clinical Research Center for Accurate Diagnosis and Treatment of High-incidence Sexually Transmitted Diseases, The First Affiliated Hospital, Hengyang Medical School, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918); University of South China, Institute of Pathogenic Biology and Key Laboratory of Special Pathogen Prevention and Control of Hunan Province, Department of Clinical Laboratory Medicine, The First Affiliated Hospital, Hengyang Medical College, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918) 
 University of South China, MOE Key Lab of Rare Pediatric Diseases&Institute of Pathogenic Biology and Key Laboratory of Special Pathogen Prevention and Control of Hunan Province, Hengyang Medical College, Hengyang City, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918); University of South China, Department of Clinical Laboratory Medicine, Institution of Microbiology and Infectious Diseases, Hunan Province Clinical Research Center for Accurate Diagnosis and Treatment of High-incidence Sexually Transmitted Diseases, The First Affiliated Hospital, Hengyang Medical School, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918); Changsha Central Hospital, Department of Clinical Laboratory Medicine, Changsha, People’s Republic of China (GRID:grid.452210.0); University of South China, School of Public Health, Hengyang Medical School, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918); University of South China, Institute of Pathogenic Biology and Key Laboratory of Special Pathogen Prevention and Control of Hunan Province, Department of Clinical Laboratory Medicine, The First Affiliated Hospital, Hengyang Medical College, Hengyang, People’s Republic of China (GRID:grid.412017.1) (ISNI:0000 0001 0266 8918) 
Publication title
AMB Express; Heidelberg
Volume
15
Issue
1
Pages
126
Publication year
2025
Publication date
Dec 2025
Publisher
Springer Nature B.V.
Place of publication
Heidelberg
Country of publication
Netherlands
Publication subject
e-ISSN
21910855
Source type
Scholarly Journal
Language of publication
English
Document type
Journal Article
Publication history
 
 
Online publication date
2025-08-25
Milestone dates
2025-08-20 (Registration); 2025-07-02 (Received); 2025-08-20 (Accepted)
Publication history
 
 
   First posting date
25 Aug 2025
ProQuest document ID
3243584658
Document URL
https://www.proquest.com/scholarly-journals/i-treponema-pallidum-inhibits-cd4-t-cell/docview/3243584658/se-2?accountid=208611
Copyright
© The Author(s) 2025. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.
Last updated
2025-08-26
Database
ProQuest One Academic