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Abstract
According to the breakpoint on EML4 (from exon 2 to exon 20), 13 variants of the EML4-ALK fusion gene have been found [7]. Prior to treatment, it is necessary to assess ALK rearrangement status to determine the appropriate therapeutic regimen. Because the underlying mechanism of the partial response to targeted therapy is unknown, we will continue to investigate this mechanism in patients with NSCLC in the future.
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