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Abstract
Excessive mobilization of extracellular potassium into cells. a. Alkalosis: She did not have a history of diuretic use or aldosteronism-related diseases. [...]hypokalemia caused by alkalosis was excluded. b. Increased β-adrenal activity: The thyroid function of this patient exhibited normal, and her symptoms did not conform to the clinical features of periodic paralysis. d. Significantly increased erythrocyte production: this condition is commonly observed after the use of folic acid, Vitamin B12, and granulocyte-macrophage colony-stimulating factor. The patient did not have a history of using the above drugs; In addition, this patient did not have chloroquine and barium poisoning and did not have a history of encountering a low-temperature environment. [...]hypokalemia caused by the excessive mobilization of extracellular potassium into cells was excluded. 3. [...]this patient exhibited low blood pressure. [...]hypokalemia caused by diseases related to mineralocorticoid increase was not considered. (2) Hereditary diseases or secondary injury of renal tubular dysfunction. [...]the common identifications include the following: According to clinical presentations, the major symptoms include the following: (1) Hypokalemia (2) hyperkaluria (3) metabolic alkalosis (4) hyperreninemia (5) hyperaldosteronemia (6) exogenous vasopressin insensitivity (7) juxtaglomerular hyperplasia (8) hypochloremia (9) normal blood pressure, and (10) clear family history. [2] Except for hypokalemia, this patient did not exhibit the above clinical presentation. [...]the possibility of LS was low. [...]GS was highly suspected. Department of Nephrology, Chinese People's Liberation Army General Hospital, Chinese People's Liberation Army Institute of Nephrology, State Key Laboratory of Kidney Diseases, National Clinical Research Center for Kidney Diseases, Beijing Key Laboratory of...