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Received Jun 11, 2017; Accepted Jul 6, 2017
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1. Introduction
Lymphedema is a type of edema caused by a failure in the formation and/or drainage of lymph that results in an accumulation of macromolecules and fluids in the interstitial space [1]. Lymphedema is classified as primary and secondary etiopathogenesis. In primary lymphedema, alterations exist at birth and in secondary disease; the patient suffers lesions to the lymphatics during life due to inflammatory or infectious processes, neoplasia, or traumas [1, 2]. Furthermore, the clinical classification is divided into four stages, where stage 0 is subclinical and in stage I there is no edema in the morning, however, during the day edema develops with a lessening of the swelling on resting with the legs elevated. Stage II is characterized by edema in the morning, with no improvement with rest and worsening during the day. Finally, stage III is the most advanced phase of lymphedema, popularly known as elephantiasis, which is identified by deformities of the affected region [1, 3].
Lipedema is an inherited condition that is characterized by bilateral and symmetrical enlargement of the lower limbs. It can be accompanied by symptoms such as hyperalgesia and cutaneous hypothermia. When lipedema progresses to lymphedema, it is called lipolymphedema. On the other hand, idiopathic cyclic edema is a clinical condition related to vessel hypermeability, associated with increased interstitial fluid and characterized by...