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© 2013 . This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.

Abstract

Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects were not significantly more than that in the control group (ptrend = 0.2752; Figure 2A); and the number of risk alleles did not increase the risk of IFH (1.053 [0.963-1.150] per allele, p = 0.2564; Figure 2C).

Details

Title
Correction: Association of Genetic Variants with Isolated Fasting Hyperglycaemia and Isolated Postprandial Hyperglycaemia in a Han Chinese Population
Author
Kong, Xiaomu; Hong, Jing; Chen, Ying; Chen, Li; Zhao, Zhigang; Li, Qiang; Ge, Jiapu; Chen, Gang; Guo, Xiaohui; Lu, Juming; Weng, Jianping; Jia, Weiping; Ji, Linong; Xiao, Jianzhong; Shan, Zhongyan; Liu, Jie; Tian, Haoming; Ji, Qiuhe; Zhu, Dalong; Zhou, Zhiguang; Shan, Guangliang; Yang, Wenying
Section
Correction
Publication year
2013
Publication date
Aug 2013
Publisher
Public Library of Science
e-ISSN
19326203
Source type
Scholarly Journal
Language of publication
English
ProQuest document ID
1974811254
Copyright
© 2013 . This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.