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Abstract
Late anterior crowding of teeth has been associated with the anterior component of force (ACF) developed during biting. Possible physiologic mechanisms countering ACF, including the presence of a posterior component of force (PCF), are hypothesized. In this self-controlled study, 60 subjects aged 27.05±3.9 years were examined for ACF and PCF that were calculated as the change in tightness of a mandibular dental contact points from non-biting to biting state. Both ACF and PCF were found to develop simultaneously. However, the PCF was 4-7 folds smaller than the ACF (p <0.001). The ACF progressively declined by 10-20 folds (p <0.001) from the posterior to anterior dentition. The lateral incisor-canine contact point had the greatest ACF decline (63-74%). ACF effect on the anterior dentition is counteracted by a protective mechanism consisted of PCF, progressive dissipation of ACF, and canine blockage.





